signs of aging by disabling a mutation that damages cells [19659004] When they turned off this mutation, the mice regrew thick fur and their skin became smoother – the same as the healthy mouse of the same age.
mitochondria in the cell is also linked to age-related diseases such as cardiovascular disease, diabetes, cancer and neurological diseases.Scientists used the doxycycline antibiotic, often used to treat bacterial infections, including acne and pneumonia.
After four weeks, they showed gray, sparse, aging and lethargic hair.
Their skin became thicker, more inflamed and their hair follicles stopped regenerating.
002] But when the cell-damaging mutation was extinguished, by stopping the antibiotics, the mice returned to their anterior appearance.
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The main author, Keshav Singh, professor of genetics at the university, said: "To our knowledge, this observation is unprecedented.
"This mouse model should offer an unprecedented opportunity for the development of preventive and therapeutic drugs to develop nt strategies to increase mitochondrial functions for the treatment of cutaneous and capillary pathology badociated with aging and other human diseases in which mitochondrial dysfunction plays an important role.
"It suggests that the epigenetic mechanisms underlying the mitochondria in the nucleus must In other words, research suggests that mitochondria play an important role in the aging process and should be further investigated to determine new ways to reverse aging
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