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03:00 p
Wednesday, February 13, 2019
Masrawy-
A joint research team from the University of Washington's School of Medicine and the German Center for Neurodegenerative Neurology in Germany has developed a simple blood test to predict that a person is at risk of suffering from Alzheimer's disease before the onset of the disease.
The new test, which detects a type of protein in a study published yesterday, published the journal Nature Medicine, and the protein it detects is called "light neurofilament polypeptide", a structural protein that is part of the internal skeleton of nerve cells. . And when neurons in the brain are damaged or die, the protein infiltrates into the cerebrospinal fluid that engulfs the brain and spinal cord and from there into the blood, citing the site of the "Al Bayan" newspaper of the Emirates United Arab Emirates.
Previous studies have shown that the discovery of elevated levels of this protein in the patient's cerebrospinal fluid convincingly demonstrates that some of his brain cells have been damaged and are at risk of developing Alzheimer's and Alzheimer's disease. other neurological diseases, but obtaining this liquid for diagnosis requires a procedure. "Lumbar puncture" or "lumbar puncture" is a process in which cerebrospinal fluid is removed from the spinal canal for diagnostic purposes.
The new study found that this protein increased in people exposed to the disease before the emergence of signs of the disease until age 16, as mentioned by Mathias Joker, a "Middle East" researcher in neuroscience at the German Center for Neurology of Tübingen.
"This test will not take much time, it will be cheaper and soon available." There are compounds very similar to those used by the authors to test blood protein levels.They are designed to predict the risk of injury. However, it has not yet been approved by the United States Food and Drug Administration (FDA). "We are currently testing new compounds and determining the amount of protein in the blood in which we can predict a risk. close."
The team came to this conclusion after studying very early 400 people from families with rare genetic variants at the origin of Alzheimer's disease.
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