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The “Covid-19” epidemic has pushed the obesity crisis back into the spotlight, revealing that obesity is no longer a disease that only harms long-term, but can have very devastating effects.
New studies and information confirm doctors’ suspicions that this virus benefits from a disease that the current healthcare system in the United States cannot control.
The Centers for Disease Control and Prevention reported that 73% of medical staff hospitalized with “Covid-19” suffer from obesity. In addition, a recent study found that obesity may interfere with the effectiveness of the Covid-19 vaccine.
Kate Farney, an obesity specialist who works on the front lines of obesity in primary care at the University of Virginia Health System, revealed that she often finds herself warning patients that obesity could shorten years of their life. Today more than ever, this warning is verifiable.
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At first, doctors thought that obesity only increased your risk of developing “Covid-19” disease, not your risk of getting infected in the first place.
Now, the latest analysis shows that obesity doesn’t just increase your risk of getting sick and dying from ‘Covid-19’. Obesity increases your risk of developing the infection in the first place.
And in March 2020, observational studies indicated that high blood pressure, diabetes and coronary heart disease are the other most common conditions – or co-morbidities – in more severe patients with Covid-19.
But the editors of a magazine Obesity They were the first to sound the alarm for the first time on April 1, 2020, that obesity is likely to be an independent risk factor for the most serious effects of ‘Covid-19’ infection.
In addition, two studies have shown that patients with “Covid-19” and obesity are more likely to die on days 21 and 45, compared to patients with a normal body mass index.
A study published in September 2020 reported high rates of obesity in patients with “Covid-19”, who are critically ill and need to be intubated.
And it has become increasingly clear from these and other studies that obese people face a clear and current danger.
Obesity is an interesting disease that many doctors talk about, often frustrated because their patients cannot prevent or reverse it with a simplified treatment plan. It is also a disease that causes physical problems, such as sleep apnea and joint pain.
Excess adipose tissue, which stores fat, creates mechanical stress in obese patients, which limits their ability to absorb and release a breath of air.
Obesity leads to an increase in adipose tissue, or what we colloquially call “fats.” Over the years, scientists have learned that fatty tissue is inherently harmful.
You could say that fatty tissue functions as an endocrine organ on its own. It secretes many hormones and molecules that lead to chronic inflammatory conditions in obese patients.
When the body is in a constant state of low-grade inflammation, it releases cytokines, which are proteins that fight inflammation and keep the body in a disease-fighting alert state.
However, when chronically released, an imbalance can develop and cause injury to the body.
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And “Covid-19” causes the body to cause another cell fire (defensive storm). And when an obese person suffers from “Covid-19”, two small fires of cytokines combine, igniting the fires of inflammation that harms the lungs more than patients with a normal body mass index.
In addition, this chronic inflammatory disease can lead to what is called endothelial dysfunction. In this condition, instead of opening, the blood vessels close and constrict, reducing oxygen to the tissues.
In addition, the increased body fat may contain more of the ACE-2 enzyme, which allows the coronavirus to invade cells and start damaging them. A recent study showed an association between the increase in ACE-2 in fatty tissue instead of lung tissue.
This finding reinforces the hypothesis that obesity plays a major role in the more serious “Covid-19” infection.
So in theory, if you have more fatty tissue, the virus can attach itself and invade more cells, causing increased viral loads that stay on for longer, which could worsen the infection and prolong the recovery period.
Angiotensin-2 converting enzyme may be helpful in fighting inflammation, but if it’s linked to “Covid-19” it won’t help.
It seems the time has come to educate physicians and provide them with the resources to tackle obesity.
Source: ScienceAlert
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