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As the worlds of beauty and advanced science intermingle more and more, the role our genes play in healthy, youthful skin has never been so clear, the discoveries formerly reserved for cancer research and autoimmune diseases. about the aging face. The meticulous DIY of the epigenome, also known as the control panel for our genes, has resulted in a number of significant recent findings but perhaps not as surreal as those reported in a recent Newsweek report [19659002]. A new study conducted by the University of Alabama at Birmingham on mitochondrial depletion in mice has found a way to disable the mutation of the gene that causes aging, by radically reversing wrinkled skin and the loss of hair.
the use to survive is created by mitochondria, mitochondrial dysfunction is intrinsically related to aging in humans. When scientists activated a mutation in mice that resulted in the decline of their mitochondrial function, they began to lose their hair, showing gray hair and developing wrinkles (the product of intrinsic and extrinsic aging) in four to eight weeks . But when the mutation of the gene was deactivated and the normal mitochondrial function was restored, the mice lost their wrinkles and found their juvenile coat. "To our knowledge, this observation is unprecedented," says Keshav Singh, one of the principal investigators. "It suggests that the epigenetic mechanisms underlying nuclear mitochondria must play an important role in restoring the normal phenotype of skin and hair."
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