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According to one study, genes tend to interact with the obesogens present in the environment, which can be a double blow for people who are genetically connected to become obese.
Obesogens are chemicals – pesticides, phthalates, drugs – that affect bodily functions and make body.
For the study, published in the journal BMJ, Norwegian researchers used data from the Nord-Trøndelag health study. Of a total of 118,959 people who participated in the study, 67,305 were included in analyzes of the association between genetic predisposition and body mass index (BMI).
The height and weight of those aged 13 to 80 years were measured several times between 1963 and 2008.
The result showed that there was an overall increase in obesity over the years. But, in people genetically predisposed to obesity, the increase was higher. Genetic predisposition was identified by participants' polygenic risk scores.
They were divided into five equal groups (the top five being the most likely to have a high BMI and the last five the least). Their BMI differed considerably between the highest and lowest groups in each decade.
The difference increased between the 1960s and the 2000s: for example, in the 1960s, the 35-year-old genetically predisposed were on average 3.9 kilograms heavier than their genetically protected peers.
But the 35-year-old genetically predisposed people were generating more than 6.8 kg in 2008, said Maria Brandkvist, the lead author of the study, while explaining that the environment is a major factor in l & # 39; obesity.
In addition, both groups gained an additional 7.1 kg due to the obesogenic environment.
Rising levels of environmental toxins is the main reason for this increase, the researchers said.
"Obese people are often stigmatized for having unhealthy lifestyle choices. Recognizing the importance of the obesogenic environment and its amplification of our genetic differences can contribute to the destigmatization of obesity, "said Brandkvist.
"Maybe it's time to change our outlook on the individual and on a healthier society," Brandkvist added.
The study has policy implications being developed to combat the epidemic of obesity. Controlling the presence of obesogens in the environment would work, while the action would also reduce other diseases such as cancer.
Researchers have long debated whether genes or the environment caused cancer. However, a Swedish study of twins showed that environmental factors caused about twice as many cancers as hereditary factors.
The risk of developing breast cancer, for example, is less than nine percent if your sister is affected, according to the study.
It was a great respite for those with a family history of cancer who feared becoming victims of cancer. He has also supported more research on the links between disease and the environment.
Obesogens include chemicals such as:
- Monosodium Glutamate – commonly known as ajinomoto
- Clozapine, an antipsychotic, genistein, found in soy products such as milk and baby food,
- Bisphenol A (BPA): Used in plastic baby bottles and baby bottles and in the sealing of cans.
- Arsenic: chemical present in rocks and soil, infiltrating into groundwater and accumulating in crops,
- Nicotine: an addictive chemical found in tobacco,
- Dichlorodiphenyltrichloroethane or popular insecticide with DDT.
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